Respiratory distress syndrome, neonatal.

Respiratory distress syndrome, neonatal.
respiratory failure in newborns is very dangerous disease leading cause of death for premature babies. Premature baby’s body is very weak and more prone to disease. Therefore, the premature baby needs care more carefully.

Children with respiratory distress symptoms at birth or a few hours after birth: rapid breathing (more than 60 beats/minute), grunting, chest concave withdrawal, cyanosis (lack of blood oxygen), nasal flaring is also a sign respiratory distress signal. Babies gradually exhausted, leading to slowed breathing and apnea.

In alveolar cells, a type of gas-exchange role, Category 2 and reserves specialized synthetic surfactant. Two types of cells differentiated only start from a 24th gestational week, mostly at about week 34. Therefore, premature infants before 34 weeks of age are more at risk of atelectasis leading to respiratory failure due to insufficient surfactant substances.

In addition to the lack of surfactant, another reason that babies are prone to respiratory depression structure has not fully formed lungs (alveoli only began to form from the 30th week). Consequently, gas exchange inefficient because it happens mostly in the bronchioles. In premature infants, the respiratory muscles have not fully developed, the soft chest should easily collapsible lungs.

Respiratory distress syndrome, neonatal

Respiratory distress syndrome, neonatal.

Other causes leading to respiratory failure in newborns .
– Genetics: In a mother ever preterm birth in respiratory failure, this risk in later births up to 90%. Based neonatal respiratory failure at the high percentage in whites; vulnerable young men and young women are more severe (as androgen-inhibiting the production of surfactant).

– Mother with diabetes: Mother’s high blood sugar levels cause insulin levels higher than a normal pregnancy. Insulin inhibits the alveolar cells mature surfactant production, causing a birth rate of respiratory failure diabetic mothers 6 times higher than other women.

– Injury perinatal: Asphyxia and hemorrhage before birth increases the risk of respiratory depression. The shortage of blood and oxygen the blood acidic, blood pressure will inhibit the synthesis of surfactant, alveolar cell destruction specialized duty gas exchange and pulmonary capillaries, leading to pulmonary edema, surfactant dysfunction. In addition, children with hypothermia at birth also cause blood oxygen deprivation and blood acid, inhibit surfactant function.

– Born surgery: The labor release of catecholamine hormones and steroid groups, stimulates the production and release of surfactant, which leads to increased lung fluid reabsorption through the lymphatic system of the lungs.